[1] BRADFORD K C, WILKINS H, HAO P, et al. Dynamic human MutSalpha-MutLalpha complexes compact mismatched DNA [J]. Proc Natl Acad Sci U S A, 2020, 117(28): 16302 − 16312. doi:  10.1073/pnas.1918519117
[2] CHATTERJEE N, WALKER G C. Mechanisms of DNA damage, repair, and mutagenesis [J]. Environ Mol Mutagen, 2017, 58(5): 235 − 263.
[3] CASTANEDA-GARCIA A, PRIETO A I, RODRIGUEZ-BELTRAN J, et al. A non-canonical mismatch repair pathway in prokaryotes [J]. Nat Commun, 2017, 8(14246): 14246.
[4] BAN C, JUNOP M, YANG W. Transformation of MutL by ATP binding and hydrolysis: a switch in DNA mismatch repair [J]. Cell, 1999, 97(1): 85 − 97.
[5] HALL M C, SHCHERBAKOVA P V, KUNKEL T A. Differential ATP binding and intrinsic ATP hydrolysis by amino-terminal domains of the yeast Mlh1 and Pms1 proteins [J]. J Biol Chem, 2002, 277(5): 3673 − 3679.
[6] GROOTHUIZEN F S, SIXMA T K. The conserved molecular machinery in DNA mismatch repair enzyme structures [J]. DNA Repair (Amst), 2016, 38: 14 − 23.
[7] QIU R, DEROCCO V C, HARRIS C, et al. Large conformational changes in MutS during DNA scanning, mismatch recognition and repair signalling [J]. EMBO J, 2012, 31(11): 2528 − 40. doi:  10.1038/emboj.2012.95
[8] GROOTHUIZEN F S, WINKLER I, CRISTOVAO M, et al. MutS/MutL crystal structure reveals that the MutS sliding clamp loads MutL onto DNA [J]. Elife, 2015, 4: e06744. doi:  10.7554/eLife.06744
[9] DUPES N M, WALSH B W, KLOCKO A D, et al. , Mutations in the Bacillus subtilis beta clamp that separate its roles in DNA replication from mismatch repair [J]. J Bacteriol, 2010, 192(13): 345234 − 63.
[10] SIMMONS L A, DAVIES B W, GROSSMAN A D, et al. , Beta clamp directs localization of mismatch repair in Bacillus subtilis [J]. Mol Cell, 2008, 29(3): 291 − 301. doi:  10.1016/j.molcel.2007.10.036
[11] PILLON M C, LORENOWICZ J J, UCKELMANN M, et al. Structure of the endonuclease domain of MutL: unlicensed to cut [J]. Mol Cell, 2010, 39(1): 145 − 51. doi:  10.1016/j.molcel.2010.06.027
[12] PILLON M C, MILLER J H, GUARNE A. The endonuclease domain of MutL interacts with the beta sliding clamp [J]. DNA Repair (Amst), 2011, 10(1): 87 − 93.
[13] SHEN M, ZHANG H, SHEN W, et al. , Pseudomonas aeruginosa MutL promotes large chromosomal deletions through non-homologous end joining to prevent bacteriophage predation [J]. Nucleic Acids Res, 2018, 46(9): 4505 − 4514. doi:  10.1093/nar/gky160
[14] 白璐, 吴利英, 吕艳红, 等. MLH1、PRMT5在卵巢癌组织中的表达及意义[J]. 生殖医学杂志, 2020, 29(10): 1356 − 1361.
[15] 陈芳, 王敏, 赵声远, 等. 鲍曼不动杆菌错配修复基因mutL的序列及其系统进化关系分析[J]. 中国微生态学杂志, 2017, 29(5): 514 − 520.
[16] 赵尔樱, 唐乐, 周玉洁, 等. 细菌基因组稳定性和变异性之间动态平衡的自发调控: 遗传开关的作用及其分子机制[J]. 中国科学, 2013, 43(15): 361 − 367.
[17] SWING J. Reclassification of the causal agents of bacterial blight (Xanthomonas campestris pv. oryzae) and bacterial leaf streak (Xanthomonas campestris pv. oryzicola) of rice as pathovars of Xanthomonas oryzae (ex Ishiyama 1922) sp. nov. nom. rev [J]. Int J System Evol Microbiol, 1990, 40: 309 − 311.
[18] SHEN Y, RONALD P. Molecular determinants of disease and resistance in interactions of Xanthomonas oryzae pv oryzae and rice [J]. Microbes and Infection, 2002, 4: 1361 − 1367. doi:  10.1016/S1286-4579(02)00004-7
[19] LI C X, LIU S Y, LIU P Y, et al. The yebN leader rNA acts as a Mn2+ sensor required for the interaction of Xanthomonas oryzae and rice [J]. Mol Plant-Microbe Ineract, 2018, 31: 932 − 939. doi:  10.1094/MPMI-02-18-0043-R
[20] LI T, ZHAN Z, LIN Y, et al. Biosynthesis of amino acids in Xanthomonas oryzae pv. oryzae is essential to its pathogenicity [J]. Microorganisms, 2019, 7(12): 693. doi:  10.3390/microorganisms7120693
[21] VERMA G, MONDAL K K, KULSHRESHTHA A, et al. XopR T3SS-effector of Xanthomonas oryzae pv. oryzae suppresses cell death-mediated plant defense response during bacterial blight development in rice [J]. 3 Biotech, 2019, 9: 272.
[22] LEE H, POPODI E, TANG H, et al. Rate and molecular spectrum of spontaneous mutations in the bacterium Escherichia coli as determined by whole-genome sequencing [J]. Proc Natl Acad Sci U S A, 2012, 109(41): e2774 − 2783.
[23] KUCUKYILDIRIM S, LONG H, SUNG W, et al. The rate and spectrum of spontaneous mutations in mycobacterium smegmatis, a bacterium naturally devoid of the postreplicative mismatch repair pathway [J]. G3 (Bethesda), 2016, 6(7): 2157 − 2163. doi:  10.1534/g3.116.030130
[24] LONG H, KUCUKYILDIRIM S, SUNG W, et al. Background mutational features of the radiation-resistant bacterium Deinococcus radiodurans [J]. Mol Biol Evol, 2015, 32(9): 2383 − 2392. doi:  10.1093/molbev/msv119